[1] Maddineni G, Xie JJ, Brahmbhatt B, et al. Diet and carcinogenesis of gastric cancer [J]. Curr Opin Gastroenterol, 2022, 38(6): 588-591. DOI: 10.1097/MOG.0000000000000875.
[2] McNamara KM, Gobert AP, Wilson KT. The role of polyamines in gastric cancer [J]. Oncogene, 2021, 40(26): 4399-4412. DOI: 10.1038/s41388-021-01862-x.
[3] Machlowska J, Baj J, Sitarz M, et al. Gastric cancer: epidemiology, risk factors, classification, genomic characteristics and treatment strategies [J]. Int J Mol Sci, 2020, 21(11): 4012. DOI: 10.3390/ijms21114012.
[4] Sung H, Ferlay J, Siegel RL, et al. Global cancer statistics 2020: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries [J]. CA Cancer J Clin, 2021, 71(3): 209-249. DOI: 10.3322/caac.21660.
[5] Li X, Zhu X, Wang Y, et al. Prognostic value and association of Lauren classification with VEGF and VEGFR-2 expression in gastric cancer [J]. Oncol Lett, 2019, 18(5): 4891-4899. DOI: 10.3892/ol.2019.10820.
[6] Correa P. Human gastric carcinogenesis: a multistep and multifactorial process--First American Cancer Society Award Lecture on Cancer Epidemiology and Prevention [J]. Cancer Res, 1992, 52(24): 6735-6740.
[7] Shao L, Li P, Ye J, et al. Risk of gastric cancer among patients with gastric intestinal metaplasia [J]. Int J Cancer, 2018, 143(7): 1671-1677. DOI: 10.1002/ijc.31571.
[8] Sugano K, Moss SF, Kuipers EJ. Gastric intestinal metaplasia: real culprit or innocent bystander as a precancerous condition for gastric cancer? [J]. Gastroenterology, 2023, 165(6):1352-1366.e1. DOI: 10.1053/j.gastro.2023.08.028.
[9] Sipponen P, Hyvärinen H, Seppälä K, et al. Review article: pathogenesis of the transformation from gastritis to malignancy [J]. Aliment Pharmacol Ther, 1998, 12 Suppl 1: 61-71. DOI: 10.1111/j.1365-2036.1998.00005.x.
[10] 中国抗癌协会胃癌专业委员会,中国医师协会外科医师分会上消化道外科医师委员会,中国人群健康风险管理协作组-胃癌专业组. 中国人群胃癌风险管理公众指南(2023版)[J]. 中华医学杂志,2023,103(36):2837-2849. DOI:10.3760/cma.j.cn112137-20230608-00968.
[11] Bockerstett KA, Lewis SA, Noto CN, et al. Single-cell transcriptional analyses identify lineage-specific epithelial responses to inflammation and metaplastic development in the gastric corpus [J]. Gastroenterology, 2020, 159(6): 2116-2129.e4. DOI: 10.1053/j.gastro. 2020.08.027.
[12] El-Zimaity HM, Ota H, Graham DY, et al. Patterns of gastric atrophy in intestinal type gastric carcinoma [J]. Cancer, 2002, 94(5): 1428-1436. DOI: 10.1002/cncr.10375.
[13] Coker OO, Dai Z, Nie Y, et al. Mucosal microbiome dysbiosis in gastric carcinogenesis [J]. Gut, 2018, 67(6): 1024-1032. DOI: 10.1136/gutjnl-2017-314281.
[14] Vinasco K, Mitchell HM, Kaakoush NO, et al. Microbial carcinogenesis: lactic acid bacteria in gastric cancer [J]. Biochim Biophys Acta Rev Cancer, 2019, 1872(2): 188309. DOI: 10.1016/j.bbcan.2019.07.004.
[15] Yu C, Su Z, Li Y, et al. Dysbiosis of gut microbiota is associated with gastric carcinogenesis in rats [J]. Biomed Pharmacother, 2020, 126: 110036. DOI: 10.1016/j.biopha.2020.110036.
[16] Sivasubramanian BP, Dave M, Panchal V, et al. Comprehensive review of red meat consumption and the risk of cancer [J]. Cureus, 2023, 15(9): e45324. DOI: 10.7759/cureus.45324.
[17] Kim SR, Kim K, Lee SA, et al. Effect of red, processed, and white meat consumption on the risk of gastric cancer: an overall and dose-response meta-analysis [J]. Nutrients, 2019, 11(4): 826. DOI: 10.3390/nu11040826.
[18] Collatuzzo G, Etemadi A, Sotoudeh M, et al. Meat consumption and risk of esophageal and gastric cancer in the Golestan Cohort Study, Iran [J]. Int J Cancer, 2022, 151(7): 1005-1012. DOI: 10.1002/ijc.34056.
[19] Pegg AE. Mammalian polyamine metabolism and function [J]. IUBMB Life, 2009, 61(9): 880-894. DOI: 10.1002/iub.230.
[20] Casero RA, Murray Stewart T, Pegg AE. Polyamine metabolism and cancer: treatments, challenges and opportunities [J]. Nat Rev Cancer, 2018, 18(11): 681-695. DOI: 10.1038/s41568-018-0050-3.
[21] Brzozowski T, Konturek PC, Konturek SJ, et al. Involvement of ornithine decarboxylase and polyamines in epidermal growth factor-induced recovery of gastric mucosa from gastric lesions provoked by stress [J]. Regul Pept, 1998, 74(2-3): 73-84. DOI: 10.1016/s0167-0115(98)00020-2.
[22] Miao XP, Li JS, Li HY, et al. Expression of ornithine decarboxylase in precancerous and cancerous gastric lesions [J]. World J Gastroenterol, 2007, 13(20): 2867-2871. DOI: 10.3748/wjg.v13.i20.2867.
[23] Brooks WH. Increased polyamines alter chromatin and stabilize autoantigens in autoimmune diseases [J]. Front Immunol, 2013, 4: 91. DOI: 10.3389/fimmu.2013.00091.
[24] Muñoz-Esparza NC, Latorre-Moratalla ML, Comas-Basté O, et al. Polyamines in food [J]. Front Nutr, 2019, 6: 108. DOI: 10.3389/fnut.2019.00108.
[25] Ding N, Cheng Y, Liu H, et al. Fusobacterium nucleatum infection induces malignant proliferation of esophageal squamous cell carcinoma cell by putrescine production [J]. Microbiol Spectr, 2023, 11(2): e0275922. DOI: 10.1128/spectrum.02759-22.
[26] 李建生,牛正先,周天星,等. 消化道肿瘤患者鸟氨酸脱羧酶和腐胺测定的临床意义[J]. 医师进修杂志,2000,23(11):20-22. DOI: 10.3760/cma.j.issn.1673-4904.2000.11.009.
[27] 吴新新,郭永军. 鸟氨酸脱羧酶与结直肠癌关系的研究进展[J]. 河南医学研究,2015(8):68-70. DOI:10.3969/j.issn. 1004-437X.2015.08.033.
[28] 张栓虎. 中国居民饮食习惯与胃癌发病关系的Meta分析[J]. 现代预防医学,2008,35(2):216-219. DOI: 10.3969/j.issn.1003-8507.2008.02.004.
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