[1] Caturegli P, De Remigis A, Rose NR. Hashimoto thyroiditis: clinical and diagnostic criteria [J]. Autoimmun Rev, 2014, 13(4-5): 391-397. DOI: 10.1016/j.autrev.2014.01.007.
[2] Lillevang-Johansen M, Abrahamsen B, Jørgensen HL, et al. Duration of over- and under-treatment of hypothyroidism is associated with increased cardiovascular risk [J]. Eur J Endocrinol, 2019, 180(6): 407-416. DOI: 10.1530/EJE-19-0006.
[3] Hasham A, Tomer Y. Genetic and epigenetic mechanisms in thyroid autoimmunity [J]. Immunol Res, 2012, 54(1-3): 204-213. DOI: 10.1007/s12026-012-8302-x.
[4] Zhang QY, Ye XP, et al. Lymphocyte infiltration and thyrocyte destruction are driven by stromal and immune cell components in Hashimoto's thyroiditis [J]. Nat Commun, 2022, 13(1): 775. DOI: 10.1038/s41467-022-28120-2.
[5] Zheng H, Xu J, Chu Y, et al. A global regulatory network for dysregulated gene expression and abnormal metabolic signaling in immune cells in the microenvironment of Graves' disease and Hashimoto's thyroiditis [J]. Front Immunol, 2022,13:879824. DOI: 10.3389/fimmu.2022.879824.
[6] Tomari S, Watanabe M, Inoue N, et al. The polymorphisms in the thyroid peroxidase gene were associated with the development of autoimmune thyroid disease and the serum levels of anti-thyroid peroxidase antibody [J]. Endocr J, 2017, 64(10): 1025-1032. DOI: 10.1507/endocrj.EJ17-0191.
[7] Mizuma T, Watanabe M, Inoue N, et al. Association of the polymorphisms in the gene encoding thyroglobulin with the development and prognosis of autoimmune thyroid disease [J]. Autoimmunity, 2017, 50(6): 386-392. DOI: 10.1080/08916934.2017.1344971.
[8] Bossowski A, Czarnocka B, Stasiak-Barmuta A, et al. Analysis of Fas, FasL and Caspase-8 expression in thyroid gland in young patients with immune and non-immune thyroid diseases [J]. Endokrynol Pol, 2007, 58(4):303-313.
[9] Yang Q, Jeremiah Bell J, Bhandoola A. T-cell lineage determination [J]. Immunol Rev, 2010, 238(1):12-22. DOI: 10.1111/j.1600-065X.2010.00956.x.
[10] Druet P, Sheela R, Pelletier L. TH1 and TH2 lymphocytes in autoimmunity [J]. Adv Nephrol Necker Hosp, 1996, 25:217-241. DOI: 10.1111/j.1440-1797.1996.tb00091.x.
[11] Phenekos C, Vryonidou A, Gritzapis AD, et al. Th1 and Th2 serum cytokine profiles characterize patients with Hashimoto's thyroiditis (Th1) and Graves' disease (Th2) [J]. Neuroimmunomodulation, 2004, 11(4): 209-213. DOI: 10.1159/000078438.
[12] Ben-Skowronek I, Szewczyk L, Kulik-Rechberger B, et al. The differences in T and B cell subsets in thyroid of children with Graves' disease and Hashimoto's thyroiditis [J]. World J Pediatr, 2013, 9(3): 245-250. DOI: 10.1007/s12519-013-0398-0.
[13] Nanba T, Watanabe M, Inoue N, et al. Increases of the Th1/Th2 cell ratio in severe Hashimoto's disease and in the proportion of Th17 cells in intractable Graves' disease [J]. Thyroid, 2009, 19(5):495-501. DOI: 10.1089/thy.2008.0423.
[14] Wilson NJ, Boniface K, Chan JR, et al. Development, cytokine profile and function of human interleukin 17-producing helper T cells [J]. Nat Immunol, 2007, 8(9):950-957. DOI: 10.1038/ni1497.
[15] Li D, Cai W, Gu R, et al. Th17 cell plays a role in the pathogenesis of Hashimoto's thyroiditis in patients [J]. Clin Immunol, 2013, 149(3):411-420. DOI: 10.1016/j.clim.2013.10.001.
[16] Zake T, Skuja S, Kalere I, et al. Upregulated tissue expression of T helper (Th) 17 pathogenic interleukin (IL)-23 and IL-1β in Hashimoto's thyroiditis but not in Graves' disease [J]. Endocr J, 2019, 66(5):423-430. DOI: 10.1507/endocrj.EJ18-0396.
[17] Tanaka A, Sakaguchi S. Regulatory T cells in cancer immunotherapy [J]. Cell Res, 2017, 27(1):109-118. DOI: 10.1038/cr.2016.151.
[18] Sakaguchi S, Mikami N, Wing JB, et al. Regulatory T cells and human disease [J]. Annu Rev Immunol, 2020, 38:541-566. DOI: 10.1146/annurev-immunol-042718- 041717.
[19] Nakano A, Watanabe M, Iida T, et al. Apoptosis-induced decrease of intrathyroidal CD4(+)CD25(+) regulatory T cells in autoimmune thyroid diseases [J]. Thyroid, 2007, 17(1):25-31. DOI: 10.1089/thy.2006.0231.
[20] Chen Z, Wang Y, Ding X, et al. The proportion of peripheral blood Tregs among the CD4+ T cells of autoimmune thyroid disease patients: a meta-analysis [J]. Endocr J, 2020, 67(3):317-326. DOI: 10.1507/endocrj.EJ19-0307.
[21] Kondo M. Lymphoid and myeloid lineage commitment in multipotent hematopoietic progenitors [J]. Immunol Rev, 2010, 238(1):37-46. DOI: 10.1111/j.1600-065X. 2010.00963.x.
[22] Ramos-Leví AM, Marazuela M. Pathogenesis of thyroid autoimmune disease: the role of cellular mechanisms [J]. Endocrinol Nutr, 2016, 63(8):421-429. DOI: 10.1016/j.endonu.2016.04.003.
[23] Yu S, Qi Y, Wang H, et al. Dysfunction of CD24+CD38+ B cells in patients with Hashimoto's thyroiditis is associated with a lack of interleukin 10 [J]. Int J Biochem Cell Biol, 2017, 90:114-120. DOI: 10.1016/j.biocel.2017.08.002.
[24] Santaguida MG, Gatto I, Mangino G, et al. BREG cells in Hashimoto's thyroiditis isolated or associated to further organ-specific autoimmune diseases [J]. Clin Immunol, 2017, 184:42-47. DOI: 10.1016/j.clim.2017.04.012.
[25] Wu MY, Wang EJ, Feng D, et al. Pharmacological insights into autophagy modulation in autoimmune diseases [J]. Acta Pharm Sin B, 2021, 11(11): 3364-3378. DOI: 10.1016/j.apsb.2021.03.026.
[26] Kulig P, Musiol S, Freiberger SN, et al. IL-12 protects from psoriasiform skin inflammation [J]. Nat Commun, 2016, 7:13466. DOI: 10.1038/ncomms13466.
[27] Lubberts E. The IL-23-IL-17 axis in inflammatory arthritis [J]. Nat Rev Rheumatol, 2015, 11(7):415-29. DOI: 10.1038/nrrheum.2015.53.
[28] Lee Y, Awasthi A, Yosef N, et al. Induction and molecular signature of pathogenic TH17 cells [J]. Nat Immunol, 2012, 13(10):991-999. DOI: 10.1038/ni.2416.
[29] Zheng T, Xu C, Mao C, et al. Increased interleukin-23 in Hashimoto's thyroiditis disease induces autophagy suppression and reactive oxygen species accumulation [J]. Front Immunol, 2018, 9:96. DOI: 10.3389/fimmu.2018.00096.
[30] Xu C, Wu F, Mao C, et al. Excess iodine promotes apoptosis of thyroid follicular epithelial cells by inducing autophagy suppression and is associated with Hashimoto thyroiditis disease [J]. J Autoimmun, 2016, 75:50-57. DOI: 10.1016/j.jaut.2016.07.008.
[31] Senou M, Costa MJ, Massart C, et al. Role of caveolin-1 in thyroid phenotype, cell homeostasis, and hormone synthesis: in vivo study of caveolin-1 knockout mice [J]. Am J Physiol Endocrinol Metab, 2009, 297(2):E438-E451. DOI: 10.1152/ajpendo.90784.2008.
[32] Lu Q, Luo X, Mao C, et al. Caveolin-1 regulates autophagy activity in thyroid follicular cells and is involved in Hashimoto's thyroiditis disease [J]. Endocr J, 2018, 65(9):893-901. DOI: 10.1507/endocrj.EJ18-0003.
[33] Łacka K, Maciejewski A. The role of apoptosis in the etiopathogenesis of autoimmune thyroiditis [J]. Pol Merkur Lekarski, 2012, 32(188):87-92.
|