[1] Mir R, Elfaki I, Frah EAM, et al. Clinical correlations of lipid profiles with the age and gender in the coronary artery disease patients: a study of 3878 CAD patients from India [J]. Endocr Metab Immune Disord Drug Targets, 2022, 22(4): 440-452. DOI: 10.2174/1871530322666220304110306.
[2] Geller DS, Farhi A, Pinkerton N, et al. Activating mineralocorticoid receptor mutation in hypertension exacerbated by pregnancy [J]. Science, 2000, 289(5476):119-123. DOI: 10.1126/science.289.5476.119.
[3] Sangha GS, Goergen CJ, Prior SJ, et al. Preclinical techniques to investigate exercise training in vascular pathophysiology [J]. Am J Physiol Heart Circ Physiol, 2021, 320(4): H1566-H1600. DOI: 10.1152/ajpheart. 00719.2020.
[4] Young SG, Fielding CJ. The ABCs of cholesterol efflux [J]. Nat Genet, 1999, 22(4): 316-368. DOI: 10.1038/11878.
[5] Orsó E, Broccardo C, Kaminski WE, et al. Transport of lipids from golgi to plasma membrane is defective in tangier disease patients and Abc1-deficient mice [J]. Nat Genet, 2000, 24(2): 192-196. DOI: 10.1038/72869.
[6] Gibala MJ, Little JP, Macdonald MJ, et al. Physiological adaptations to low-volume, high-intensity interval training in health and disease [J]. J Physiol, 2012, 590(5):1077-1084. DOI: 10.1113/jphysiol.2011.224725.
[7] Currie KD, Dubberley JB, McKelvie RS, et al. Low-volume, high-intensity interval training in patients with CAD [J]. Med Sci Sports Exerc, 2013, 45(8): 1436-1442. DOI: 10.1249/MSS.0b013e31828bbbd4.
[8] Tanasescu M, Leitzmann MF, Rimm EB, et al. Exercise type and intensity in relation to coronary heart disease in men [J]. JAMA, 2002, 288(16): 1994-2000. DOI: 10.1001/jama. 288.16.1994.
[9] Manson JE, Hu FB, Rich-Edwards JW, et al. A prospective study of walking as compared with vigorous exercise in the prevention of coronary heart disease in women [J]. N Engl J Med, 1999, 341(9):650-658. DOI: 10.1056/NEJM199908263410904.
[10] Taylor JL, Bonikowske AR, Olson TP. Optimizing outcomes in cardiac rehabilitation: the importance of exercise intensity [J]. Front Cardiovasc Med, 2021, 8:734278. DOI: 10.3389/fcvm.2021.734278.
[11] Dibben G, Faulkner J, Oldridge N, et al. Exercise-based cardiac rehabilitation for coronary heart disease [J]. Cochrane Database Syst Rev, 2021, 11(11): CD001800. DOI: 10.1002/14651858.CD001800.pub4.
[12] Taylor RS, Brown A, Ebrahim S, et al. Exercise-based rehabilitation for patients with coronary heart disease: systematic review and meta-analysis of randomized controlled trials [J]. Am J Med, 2004, 116(10): 682-692. DOI: 10.1016/j.amjmed.2004.01.009.
[13] Mikkelsen N, Cadarso-Suárez C, Lado-Baleato O, et al. Improvement in VO2peak predicts readmissions for cardiovascular disease and mortality in patients undergoing cardiac rehabilitation [J]. Eur J Prev Cardiol, 2020, 27(8):811-819. DOI: 10.1177/2047487319887835.
[14] Rognmo Ø, Hetland E, Helgerud J, et al. High intensity aerobic interval exercise is superior to moderate intensity exercise for increasing aerobic capacity in patients with coronary artery disease [J]. Eur J Cardiovasc Prev Rehabil, 2004, 11(3): 216-222. DOI: 10.1097/01.hjr.0000131677. 96762.0c.
[15] Zhang R, Lahens NF, Ballance HI, et al. A circadian gene expression atlas in mammals: implications for biology and medicine [J]. Proc Natl Acad Sci U S A, 2014, 111(45):16219-16224. DOI: 10.1073/pnas.1408886111.
[16] 徐晨,钱睿哲,金惠铭. 生物钟基因与心血管疾病[J]. 中国病理生理杂志,2006,22(7):1450-1453. DOI:10.3321/j.issn:1000-4718.2006.07.046.
[17] Li JJ. Circadian variation in myocardial ischemia: the possible mechanisms involving in this phenomenon [J]. Med Hypotheses, 2003, 61(2):240-243. DOI: 10.1016/s0306-9877(03)00154-3.
[18] Penev PD, Kolker DE, Zee PC, et al. Chronic circadian desynchronization decreases the survival of animals with cardiomyopathic heart disease [J]. Am J Physiol, 1998, 275(6): H2334- H2337. DOI: 10.1152/ajpheart.1998.275.6.H2334.
[19] Martino TA, Oudit GY, Herzenberg AM, et al. Circadian rhythm disorganization produces profound cardiovascular and renal disease in hamsters [J]. Am J Physiol Regul Integr Comp Physiol, 2008, 294(5): R1675- R1683. DOI: 10.1152/ajpregu.00829.2007.
[20] Laugsand LE, Strand LB, Platou C, et al. Insomnia and the risk of incident heart failure: a population study [J]. Eur Heart J, 2014, 35(21):1382-1393. DOI: 10.1093/eurheartj/eht019.
[21] Laugsand LE, Vatten LJ, Platou C, et al. Insomnia and the risk of acute myocardial infarction: a population study [J]. Circulation, 2011, 124(19): 2073-2081. DOI: 10.1161/CIRCULATIONAHA.111.025858.
[22] Davidson AJ, London B, Block GD, et al. Cardiovascular tissues contain independent circadian clocks [J]. Clin Exp Hypertens, 2005, 27(2-3): 307-311.
[23] Crnko S, Du Pré BC, Sluijter JPG, et al. Circadian rhythms and the molecular clock in cardiovascular biology and disease [J]. Nat Rev Cardiol, 2019, 16(7): 437-447. DOI: 10.1038/s41569-019-0167-4.
[24] Takeda N, Maemura K, Horie S, et al. Thrombomodulin is a clock-controlled gene in vascular endothelial cells [J]. J Biol Chem, 2007, 282(45): 32561-32567. DOI: 10.1074/jbc.M705692200.
[25] Panza JA, Epstein SE, Quyyumi AA. Circadian variation in vascular tone and its relation to alpha-sympathetic vasoconstrictor activity [J]. N Engl J Med, 1991, 325(14): 986-990. DOI: 10.1056/NEJM199110033251402.
[26] Cheng B, Anea CB, Yao L, et al. Tissue-intrinsic dysfunction of circadian clock confers transplant arteriosclerosis [J]. Proc Natl Acad Sci U S A, 2011, 108(41): 17147-17152. DOI: 10.1073/pnas.1112998108.
[27] Hermida RC, Ayala DE. Chronotherapy with the angiotensin-converting enzyme inhibitor ramipril in essential hypertension: improved blood pressure control with bedtime dosing [J]. Hypertension, 2009, 54(1): 40-46. DOI: 10.1161/HYPERTENSIONAHA.109.130203.
[28] Hermida RC, Ayala DE, Mojón A, et al. Decreasing sleep-time blood pressure determined by ambulatory monitoring reduces cardiovascular risk [J]. J Am Coll Cardiol, 2011, 58(11): 1165-1173. DOI: 10.1016/j.jacc.2011.04.043.
[29] De Giorgi A, Mallozzi Menegatti A, Fabbian F, et al. Circadian rhythms and medical diseases: does it matter when drugs are taken? [J]. Eur J Intern Med, 2013, 24(8): 698-706. DOI: 10.1016/j.ejim.2013.03.019.
[30] Manfredini R, Gallerani M, Salmi R, et al. Circadian rhythms and the heart: implications for chronotherapy of cardiovascular diseases [J]. Clin Pharmacol Ther, 1994, 56(3):244-247. DOI: 10.1038/clpt.1994.133.
[31] Watanabe Y, Halberg F, Otsuka K, et al. Toward a personalized chronotherapy of high blood pressure and a circadian overswing [J]. Clin Exp Hypertens, 2013, 35(4): 257-266. DOI: 10.3109/10641963.2013.780073.
[32] Shinagawa M, Kubo Y, Otsuka K, et al. Impact of circadian amplitude and chronotherapy: relevance to prevention and treatment of stroke [J]. Biomed Pharmacother, 2001, 55 Suppl 1:125s-132s. DOI: 10.1016/s0753-3322(01)90017-4.
[33] Wu H, Dunnett S, Ho YS, et al. The role of sleep deprivation and circadian rhythm disruption as risk factors of Alzheimer's disease [J]. Front Neuroendocrinol, 2019, 54:100764. DOI: 10.1016/j.yfrne.2019.100764.
[34] Ruan W, Yuan X, Eltzschig HK. Circadian rhythm as a therapeutic target [J]. Nat Rev Drug Discov, 2021, 20(4):287-307. DOI: 10.1038/s41573-020-00109-w.
|